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Critical Reviews™ in Eukaryotic Gene Expression
Fator do impacto: 2.156 FI de cinco anos: 2.255 SJR: 0.649 SNIP: 0.599 CiteScore™: 3

ISSN Imprimir: 1045-4403
ISSN On-line: 2162-6502

Volumes:
Volume 30, 2020 Volume 29, 2019 Volume 28, 2018 Volume 27, 2017 Volume 26, 2016 Volume 25, 2015 Volume 24, 2014 Volume 23, 2013 Volume 22, 2012 Volume 21, 2011 Volume 20, 2010 Volume 19, 2009 Volume 18, 2008 Volume 17, 2007 Volume 16, 2006 Volume 15, 2005 Volume 14, 2004 Volume 13, 2003 Volume 12, 2002 Volume 11, 2001 Volume 10, 2000 Volume 9, 1999 Volume 8, 1998 Volume 7, 1997 Volume 6, 1996 Volume 5, 1995 Volume 4, 1994

Critical Reviews™ in Eukaryotic Gene Expression

Compre a edição $169.00 Volume 10, 2000 Edição 1

DOI: 10.1615/CritRevEukarGeneExpr.v10.i1

Sumário:

Understanding "Active" Chromatin: A Historical Perspective of Chromatin Remodeling
Jocelyn E. Krebs, Craig L. Peterson
12 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i1.20
Cell Nucleus in Context
Sophie Lelievre, Mina J. Bisseil, Philippe Pujuguet
8 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i1.30
Specialized Transcription Factories within Mammalian Nuclei
Ana Pombo, Emma Jones, Francisco J. Iborra, Hiroshi Kimura, Kimihiko Sugaya, Peter R. Cook, Dean A. Jackson
10 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i1.40
Specification of Chromatin Domains and Regulation of Replication and Transcription During Development
Yegor S. Vassetzky, Jean-Marc Lemaitre, Marcel Mechali
8 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i1.50
Nuclear Matrix Targeting of Steroid Receptors: Specific Signal Sequences and Acceptor Proteins
Donald B. DeFranco, Jennifer Guerrero
6 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i1.60
The Nuclear Matrix as a Target for Viral and Cellular Oncogenes
Wolfgang Deppert
18 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i1.70
Caught in the Act: Binding of Ku and PARP to MARs Reveals Novel Aspects of Their Functional Interaction
Sanjeev Galande, Terumi Kohwi-Shigematsu
10 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i1.80
Transcriptional Augmentation: Modulation of Gene Expression by Scaffold/Matrix-Attached Regions (S/MAR Elements)
Jurgen Bode, Craig Benham, Angela Knopp, Christian Mielke
18 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i1.90
On the Relationship of Matrix Association and DNA Replication
Bruna P. Brylawski, Stephanie M. Cohen, Mania Cordeiro-Stone, Michael J. Schell, David G. Kaufman
9 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i1.100
The Nuclear Pore Complex: Structure, Function, and Dynamics
Elena M. Kiseleva, Martin W. Goldberg, Janet Cronshaw, Terence D. Allen
12 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i1.110

Critical Reviews™ in Eukaryotic Gene Expression

Breast Cancer Bone Disease. CRE Vol. 19, Issue 1. Breast Cancer Bone Disease. CRE Vol. 19, Issue 1. Cancer Treatment-Related Bone Disease
The relationship of adipocytes to osetoblasts. Vol. 19, Iss. 2, Marrow Fat and the Bone Microenvironment: Developmental, Functional, and Pathological Implications. The relationship of adipocytes to osetoblasts. Vol. 19, Iss. 2, Marrow Fat and the Bone Microenvironment: Developmental, Functional, and Pathological Implications. Marrow Fat and the Bone Microenvironment: Developmental, Functional, and Pathological Implications
Metallothionein Expression. Vol. 19, Issue 4, Human Metallothionein Expression under Normal and Pathological Conditions: Mechanisms of Gene Regulation Based on In silico Promoter Analysis. Metallothionein Expression. Vol. 19, Issue 4, Human Metallothionein Expression under Normal and Pathological Conditions: Mechanisms of Gene Regulation Based on In silico Promoter Analysis. Human Metallothionein Expression under Normal and Pathological Conditions: Mechanisms of Gene Regulation Based on In silico Promoter Analysis
Schematic structure of mT. Vol.20, Iss. 1, mTOR Signaling in Cancer Cell Motility andtTumor Metastasis. Schematic structure of mT. Vol.20, Iss. 1, mTOR Signaling in Cancer Cell Motility andtTumor Metastasis. mTOR Signaling in Cancer Cell Motility and Tumor Metastasis
Possibilities for how the c-myc proto-oncogene can be amplified in different tumor cells.Vol. 20, Iss. 3, Genome Instability in the Context of ChromatinStructure and Fragile Sites. Possibilities for how the c-myc proto-oncogene can be amplified in different tumor cells.Vol. 20, Iss. 3, Genome Instability in the Context of ChromatinStructure and Fragile Sites. Genome Instability in the Context of Chromatin Structure and Fragile Sites
Schematic representation of the tentative neuroinflammatory mechanism and its relationship to heteroossification. Vol. 20, Iss. 4, Heterotopic Ossification Has Some Nerve. Schematic representation of the tentative neuroinflammatory mechanism and its relationship to heteroossification. Vol. 20, Iss. 4, Heterotopic Ossification Has Some Nerve. Heterotopic Ossification Has Some Nerve