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Critical Reviews™ in Eukaryotic Gene Expression

Publicou 6 edições por ano

ISSN Imprimir: 1045-4403

ISSN On-line: 2162-6502

The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two preceding years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) IF: 1.6 To calculate the five year Impact Factor, citations are counted in 2017 to the previous five years and divided by the source items published in the previous five years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) 5-Year IF: 2.2 The Immediacy Index is the average number of times an article is cited in the year it is published. The journal Immediacy Index indicates how quickly articles in a journal are cited. Immediacy Index: 0.3 The Eigenfactor score, developed by Jevin West and Carl Bergstrom at the University of Washington, is a rating of the total importance of a scientific journal. Journals are rated according to the number of incoming citations, with citations from highly ranked journals weighted to make a larger contribution to the eigenfactor than those from poorly ranked journals. Eigenfactor: 0.00058 The Journal Citation Indicator (JCI) is a single measurement of the field-normalized citation impact of journals in the Web of Science Core Collection across disciplines. The key words here are that the metric is normalized and cross-disciplinary. JCI: 0.33 SJR: 0.345 SNIP: 0.46 CiteScore™:: 2.5 H-Index: 67

Indexed in

Cellular Genetics of Host Susceptibility and Resistance to Virus Infection

Volume 16, Edição 2, 2006, pp. 155-170
DOI: 10.1615/CritRevEukarGeneExpr.v16.i2.40
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RESUMO

Viruses are obligate intracellular parasites that rely upon the host cell for activities essential to their life cycles. Gene-trap mutagenesis provides a rapid, genome-wide strategy to identify candidate cellular genes required for virus replication. The candidate genes provide a starting point for mechanistic studies of cellular processes that participate in the virus life cycle and may provide targets for novel antiviral therapies.

CITADO POR
  1. Smith Todd G., Wu Xianfu, Franka Richard, E. Rupprecht Charles, Design of Future Rabies Biologics and Antiviral Drugs, in Research Advances in Rabies, 79, 2011. Crossref

  2. Lin Ping-Yuan, Liu Hung-Jen, Liao Ming-Huei, Chang Ching-Dong, Chang Chi-I, Cheng Hsueh-Ling, Lee Jeng-Woei, Shih Wen-Ling, Activation of PI 3-kinase/Akt/NF-κB and Stat3 signaling by avian reovirus S1133 in the early stages of infection results in an inflammatory response and delayed apoptosis, Virology, 400, 1, 2010. Crossref

  3. Murray James L, McDonald Natalie J, Sheng Jinsong, Shaw Michael W, Hodge Thomas W, Rubin Donald H, O'Brien William A, Smee Donald F, Inhibition of Influenza a Virus Replication by Antagonism of a PI3K-AKT-mTOR Pathway Member Identified by Gene-Trap Insertional Mutagenesis, Antiviral Chemistry and Chemotherapy, 22, 5, 2012. Crossref

  4. Friedrich Brian M, Murray James L, Li Guangyu, Sheng Jinsong, Hodge Thomas W, Rubin Donald H, O'Brien William A, Ferguson Monique R, A Functional Role for ADAM10 in Human Immunodeficiency Virus Type-1 Replication, Retrovirology, 8, 1, 2011. Crossref

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