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Journal of Environmental Pathology, Toxicology and Oncology

年間 4 号発行

ISSN 印刷: 0731-8898

ISSN オンライン: 2162-6537

The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two preceding years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) IF: 2.4 To calculate the five year Impact Factor, citations are counted in 2017 to the previous five years and divided by the source items published in the previous five years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) 5-Year IF: 2.8 The Immediacy Index is the average number of times an article is cited in the year it is published. The journal Immediacy Index indicates how quickly articles in a journal are cited. Immediacy Index: 0.5 The Eigenfactor score, developed by Jevin West and Carl Bergstrom at the University of Washington, is a rating of the total importance of a scientific journal. Journals are rated according to the number of incoming citations, with citations from highly ranked journals weighted to make a larger contribution to the eigenfactor than those from poorly ranked journals. Eigenfactor: 0.00049 The Journal Citation Indicator (JCI) is a single measurement of the field-normalized citation impact of journals in the Web of Science Core Collection across disciplines. The key words here are that the metric is normalized and cross-disciplinary. JCI: 0.59 SJR: 0.429 SNIP: 0.507 CiteScore™:: 3.9 H-Index: 49

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Cardiotoxicity and Apoptotic Activity in Subacute Endosulfan Toxicity and the Protective Effect of Vitamin C in Rabbits: A Pathological Study

巻 32, 発行 1, 2013, pp. 53-58
DOI: 10.1615/JEnvironPatholToxicolOncol.2013006476
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要約

Cardiovascular disease is one of the most significant causes of mortality in humans and animals, and its etiology is usually unknown. The aim of this study was to investigate the cardiac pathology of endosulfan toxicity and the protective effect of vitamin C in rabbits. Twenty-four rabbits were divided into 4 groups: (1) the END group was given a daily sublethal dose of endosulfan in corn oil by oral gavage for 6 weeks; (2) the END + C group received the endosulfan as well as vitamin C over the same 6-week period; (3) the OIL + C group received corn oil daily and vitamin C every other day; and (4) the OIL group received only corn oil daily. We observed microscopic hemorrhages, single-cell necrosis, inflammatory reactions, and fibrotic changes in the myocardium in the END group. Small hemorrhages and single-cell necrosis also were seen in some hearts in the END + C group, but no inflammation was observed. Caspase-3 immunoreactivity was more significant in myocardial cells in the END group compared with the others. A protective effect of vitamin C on lesions was observed in the END + C group. These results showed that endosulfan resulted in toxic changes in the hearts of rabbits, but this toxicity could be decreased with vitamin C treatment.

によって引用された
  1. Ozmen O, Mor F, Effects of vitamin C on pathology and caspase-3 activity of kidneys with subacute endosulfan toxicity, Biotechnic & Histochemistry, 90, 1, 2015. Crossref

  2. Wei Jia-Liu, Zhang Lian-Shuang, Wang Jing-Jing, Guo Fang-Zi, Li Yan-Bo, Zhou Xian-Qing, Sun Zhi-Wei, Endosulfan inducing blood hypercoagulability and endothelial cells apoptosis via the death receptor pathway in Wistar rats, Toxicology Research, 4, 5, 2015. Crossref

  3. Wei Jialiu, Zhang Lianshuang, Ren Lihua, Zhang Jin, Yu Yang, Wang Ji, Duan Junchao, Peng Cheng, Sun Zhiwei, Zhou Xianqing, Endosulfan inhibits proliferation through the Notch signaling pathway in human umbilical vein endothelial cells, Environmental Pollution, 221, 2017. Crossref

  4. Wei Jialiu, Zhang Lianshuang, Ren Lihua, Zhang Jin, Liu Jianhui, Duan Junchao, Yu Yang, Li Yanbo, Peng Cheng, Zhou Xianqing, Sun Zhiwei, Endosulfan induces cell dysfunction through cycle arrest resulting from DNA damage and DNA damage response signaling pathways, Science of The Total Environment, 589, 2017. Crossref

  5. Shi Lin-Bo, Xu Hua-Ping, Wu Yu-Jie, Li Xin, Gao Jin-Yan, Chen Hong-Bing, The effects of imidacloprid combined with endosulfan on IgE-mediated mouse bone marrow-derived mast cell degranulation and anaphylaxis, Pesticide Biochemistry and Physiology, 148, 2018. Crossref

  6. Slade Logan, Cowie Andrew, Martyniuk Chris J., Kienesberger Petra C., Pulinilkunnil Thomas, Dieldrin Augments mTOR Signaling and Regulates Genes Associated with Cardiovascular Disease in the Adult Zebrafish Heart (Danio rerio), Journal of Pharmacology and Experimental Therapeutics, 361, 3, 2017. Crossref

  7. Sun ShiTian, Ji ZhengGuo, Fu JiaRong, Wang Xi-Feng, Zhang Lian-Shuang, Endosulfan induces endothelial inflammation and dysfunction via IRE1α/NF-κB signaling pathway, Environmental Science and Pollution Research, 27, 21, 2020. Crossref

  8. Wei Jialiu, Liu Jianhui, Zhang Lianshuang, Zhu Yupeng, Li Xiangyang, Zhou Guiqing, Zhao Yanzhi, Sun Zhiwei, Zhou Xianqing, Endosulfan induces cardiotoxicity through apoptosis via unbalance of pro-survival and mitochondrial-mediated apoptotic pathways, Science of The Total Environment, 727, 2020. Crossref

  9. Rainey Nathan Earl, Moustapha Aoula, Petit Patrice Xavier, Curcumin, a Multifaceted Hormetic Agent, Mediates an Intricate Crosstalk between Mitochondrial Turnover, Autophagy, and Apoptosis, Oxidative Medicine and Cellular Longevity, 2020, 2020. Crossref

  10. Sathishkumar Palanivel, Mohan Kannan, Ganesan Abirami Ramu, Govarthanan Muthusamy, Yusoff Abdull Rahim Mohd, Gu Feng Long, Persistence, toxicological effect and ecological issues of endosulfan – A review, Journal of Hazardous Materials, 416, 2021. Crossref

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