Abonnement à la biblothèque: Guest
Critical Reviews™ in Eukaryotic Gene Expression

Publication de 6  numéros par an

ISSN Imprimer: 1045-4403

ISSN En ligne: 2162-6502

The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two preceding years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) IF: 1.6 To calculate the five year Impact Factor, citations are counted in 2017 to the previous five years and divided by the source items published in the previous five years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) 5-Year IF: 2.2 The Immediacy Index is the average number of times an article is cited in the year it is published. The journal Immediacy Index indicates how quickly articles in a journal are cited. Immediacy Index: 0.3 The Eigenfactor score, developed by Jevin West and Carl Bergstrom at the University of Washington, is a rating of the total importance of a scientific journal. Journals are rated according to the number of incoming citations, with citations from highly ranked journals weighted to make a larger contribution to the eigenfactor than those from poorly ranked journals. Eigenfactor: 0.00058 The Journal Citation Indicator (JCI) is a single measurement of the field-normalized citation impact of journals in the Web of Science Core Collection across disciplines. The key words here are that the metric is normalized and cross-disciplinary. JCI: 0.33 SJR: 0.345 SNIP: 0.46 CiteScore™:: 2.5 H-Index: 67

Indexed in

Molecular and Functional Interactions Among Monocytes/Macrophages and Smooth Muscle Cells and Their Relevance for Atherosclerosis

Volume 24, Numéro 4, 2014, pp. 341-355
DOI: 10.1615/CritRevEukaryotGeneExpr.2014012157
Get accessGet access

RÉSUMÉ

Macrophages, smooth muscle cells (SMCs), and their interactions have key roles in the pathogenesis of atherosclerotic vascular diseases. In atheroma development, the phenotype of macrophages and SMCs change, which may influence the disease progression. Accumulating data on the phenotypes exhibited by these cells within atherosclerotic lesions raise many questions regarding the mechanisms and factors that might control the transition of cell phenotype. SMCs often reside in vascular lesions in close proximity to macrophage clusters and are most likely influenced by factors released from these proinflammatory phagocytes. Moreover, macrophages may be influenced by direct contact with SMCs or soluble factors released by these cells. Macrophages may promote activation and induce proatherogenic functions of SMCs, and SMCs may modulate macrophage phenotype. Addressing the mechanisms involved in SMC−macrophage crosstalk that lead to phenotypic modulation of both cell types may provide new insight into atherogenesis and new targets for therapies of various vascular diseases.

CITÉ PAR
  1. Tan Jin-Yun, Jia Luo-Qi, Shi Wei-Hao, He Qing, Zhu Lei, Yu Bo, Rab5a-mediated autophagy regulates the phenotype and behavior of vascular smooth muscle cells, Molecular Medicine Reports, 14, 5, 2016. Crossref

  2. Jia Su-Jie, Gao Ke-Qin, Zhao Ming, Epigenetic regulation in monocyte/macrophage: A key player during atherosclerosis, Cardiovascular Therapeutics, 35, 3, 2017. Crossref

  3. Banchhor Sumit K., Londhe Narendra D., Araki Tadashi, Saba Luca, Radeva Petia, Khanna Narendra N., Suri Jasjit S., Calcium detection, its quantification, and grayscale morphology-based risk stratification using machine learning in multimodality big data coronary and carotid scans: A review, Computers in Biology and Medicine, 101, 2018. Crossref

  4. Yu Xiao-Hua, Zhang Da-Wei, Zheng Xi-Long, Tang Chao-Ke, C1q tumor necrosis factor-related protein 9 in atherosclerosis: Mechanistic insights and therapeutic potential, Atherosclerosis, 276, 2018. Crossref

  5. Zaccagnini Germana, Greco Simona, Longo Marialucia, Maimone Biagina, Voellenkle Christine, Fuschi Paola, Carrara Matteo, Creo Pasquale, Maselli Davide, Tirone Mario, Mazzone Massimiliano, Gaetano Carlo, Spinetti Gaia, Martelli Fabio, Hypoxia-induced miR-210 modulates the inflammatory response and fibrosis upon acute ischemia, Cell Death & Disease, 12, 5, 2021. Crossref

  6. Xiang Pinhao, Blanchard Valentin, Francis Gordon A., Smooth Muscle Cell—Macrophage Interactions Leading to Foam Cell Formation in Atherosclerosis: Location, Location, Location, Frontiers in Physiology, 13, 2022. Crossref

Portail numérique Bibliothèque numérique eBooks Revues Références et comptes rendus Collections Prix et politiques d'abonnement Begell House Contactez-nous Language English 中文 Русский Português German French Spain