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Critical Reviews™ in Biomedical Engineering

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ISSN Druckformat: 0278-940X

ISSN Online: 1943-619X

SJR: 0.262 SNIP: 0.372 CiteScore™:: 2.2 H-Index: 56

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Air Pollution and Cardiovascular Disease: A Review

Volumen 44, Ausgabe 5, 2016, pp. 327-346
DOI: 10.1615/CritRevBiomedEng.2017019768
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ABSTRAKT

Air pollution is comprised of different compounds and particulate matter (PM) of sizes 2.5 and 10 μm, with the former size posing the greatest danger to humans. Evidence suggests that the global rise in air pollution levels during the past century is correlated with the increased incidence of diseases of the cardiovascular system. On a global scale, 7 million individuals died as a result of the effects of air pollution in 2012. Air pollution leads to tremendous amounts of financial burden (in 2010, $16 trillion in the US and Europe) on the health-care system. The severity of effects experienced by varying populations due to air pollution can differ due to locale, length of exposure, weather conditions, residential proximity to major highways or factories, and soil aridity. Pollutants affect the heart, blood vessels, and blood at a molecular level through proinflammatory or oxidative stress response, autonomic nervous system imbalance, and the direct permeation of harmful compounds into the tissue. The dysfunction of cells and biological processes of the cardiovascular system due to PM leads to an increased prevalence of cardiovascular diseases (CVDs) such as atherosclerosis, hypertension, myocardial infarction, thrombosis, and restricted valve motion. Studies in countries such as China have shown an increase of 0.25% in ischemic heart disease (IHD) mortality and a 0.27% increase in IHD morbidity due to a 10 μg/m3 increase in PM. In a study conducted in the US, PM2.5 concentrations ranged from 9.2−22.6 μg/m3, and every 5−μg/m3 increase in PM2.5 caused coronary calcification to increase by 4.1 Agatston units/yr. Studies on traffic-related air pollution found that nonhypertensive participants residing within 100 m of major roadways experienced an increase in systolic (0.35 mmHg) and diastolic (0.22 mmHg) blood pressure as a result of increases in traffic. The progression of CVD due to pollution has been found to fluctuate within individuals based on age, gender, location of exercise, smoking, pregnancy, diabetes, preexisting cardiovascular or pulmonary diseases, and other factors. Considering the number of individuals affected by pollution on a daily basis and the burden that this places on society through the health-care system, immediate preventive measures are needed to address these problems. Increased knowledge about the widespread effects of pollution on human physiological systems should aid in remediating the problem across the globe. Biomedical engineers can have a great positive impact in developing better instrumentation to measure discrete pollutants and characterizing their harmful effects on physiological systems.

REFERENZIERT VON
  1. Singh Ram Sewak, Saini Barjinder Singh, Sunkaria Ramesh Kumar, Arrhythmia detection based on time–frequency features of heart rate variability and back-propagation neural network, Iran Journal of Computer Science, 2, 4, 2019. Crossref

  2. Ho Chia‐Chi, Tsai Ming‐Hsien, Chen Yu‐Cheng, Kuo Cheng‐Chin, Lin Pinpin, Persistent elevation of blood pressure by ambient coarse particulate matter after recovery from pulmonary inflammation in mice, Environmental Toxicology, 34, 7, 2019. Crossref

  3. Longley Ian, Tunno Brett, Somervell Elizabeth, Edwards Sam, Olivares Gustavo, Gray Sally, Coulson Guy, Cambal Leah, Roper Courtney, Chubb Lauren, Clougherty Jane E., Assessment of Spatial Variability across Multiple Pollutants in Auckland, New Zealand, International Journal of Environmental Research and Public Health, 16, 9, 2019. Crossref

  4. Song Jie, Liu Yue, Lu Mengxue, An Zhen, Lu Jianguo, Chao Ling, Zheng Liheng, Li Juan, Yao Sanqiao, Wu Weidong, Xu Dongqun, Short-term exposure to nitrogen dioxide pollution and the risk of eye and adnexa diseases in Xinxiang, China, Atmospheric Environment, 218, 2019. Crossref

  5. Lee Mi Kyeong, Xu Cheng-Jian, Carnes Megan U., Nichols Cody E., Ward James M., Kwon Sung Ok, Kim Sun-Young, Kim Woo Jin, London Stephanie J., Genome-wide DNA methylation and long-term ambient air pollution exposure in Korean adults, Clinical Epigenetics, 11, 1, 2019. Crossref

  6. Zhang Lingzhi, Valizadeh Hamed, Alipourfard Iraj, Bidares Ramtin, Aghebati-Maleki Leili, Ahmadi Majid, Epigenetic Modifications and Therapy in Chronic Obstructive Pulmonary Disease (COPD): An Update Review, COPD: Journal of Chronic Obstructive Pulmonary Disease, 17, 3, 2020. Crossref

  7. Pang Yaxian, Liu Shipeng, Yan Lina, Wang Qian, Li Lipeng, Chu Chen, Ning Jie, Zhang Boyuan, Wang Xueliang, Ma Shitao, Su Dong, Zhang Rong, Niu Yujie, Associations of long-term exposure to traffic-related air pollution with risk of valvular heart disease based on a cross-sectional study, Ecotoxicology and Environmental Safety, 209, 2021. Crossref

  8. Yarwood Stephen John, Special Issue on “New Advances in Cyclic AMP Signalling”—An Editorial Overview, Cells, 9, 10, 2020. Crossref

  9. An Farun, Liu Jiying, Lu Wanpeng, Jareemit Daranee, A review of the effect of traffic-related air pollution around schools on student health and its mitigation, Journal of Transport & Health, 23, 2021. Crossref

  10. Saini Jagriti, Dutta Maitreyee, Marques Gonçalo, Indoor Air Quality Monitoring Systems and COVID-19, in Emerging Technologies During the Era of COVID-19 Pandemic, 348, 2021. Crossref

  11. Li Zhaoyuan, Liu Yisi, Lu Tianjun, Peng Shouxin, Liu Feifei, Sun Jinhui, Xiang Hao, Acute effect of fine particulate matter on blood pressure, heart rate and related inflammation biomarkers: A panel study in healthy adults, Ecotoxicology and Environmental Safety, 228, 2021. Crossref

  12. Elbarbary Mona, Oganesyan Artem, Honda Trenton, Morgan Geoffrey, Guo Yuming, Guo Yanfei, Negin Joel, Systemic Inflammation (C-Reactive Protein) in Older Chinese Adults Is Associated with Long-Term Exposure to Ambient Air Pollution, International Journal of Environmental Research and Public Health, 18, 6, 2021. Crossref

  13. Bo Yacong, Chang Ly-yun, Guo Cui, Lin Changqing, Lau Alexis K H, Tam Tony, Yeoh Eng-Kiong, Lao Xiang Qian, Associations of Reduced Ambient PM2.5 Level With Lower Plasma Glucose Concentration and Decreased Risk of Type 2 Diabetes in Adults: A Longitudinal Cohort Study, American Journal of Epidemiology, 190, 10, 2021. Crossref

  14. Xing Ze, Zhang Shuang, Jiang Yu-Ting, Wang Xiu-Xia, Cui Hong, Association between prenatal air pollution exposure and risk of hypospadias in offspring: a systematic review and meta-analysis of observational studies, Aging, 13, 6, 2021. Crossref

  15. Ni Yu, Tracy Russell P., Cornell Elaine, Kaufman Joel D., Szpiro Adam A., Campen Matthew J., Vedal Sverre, Short-term exposure to air pollution and biomarkers of cardiovascular effect: A repeated measures study, Environmental Pollution, 279, 2021. Crossref

  16. An Farun, Liu Jiying, Lu Wanpeng, Jareemit Daranee, Comparison of exposure to traffic-related pollutants on different commuting routes to a primary school in Jinan, China, Environmental Science and Pollution Research, 29, 28, 2022. Crossref

  17. Wyatt Lauren, Kamat Gauri, Moyer Joshua, Weaver Anne M, Diaz-Sanchez David, Devlin Robert B, Di Qian, Schwartz Joel D, Cascio Wayne E, Ward-Caviness Cavin K, Associations between short-term exposure to PM2.5 and cardiomyocyte injury in myocardial infarction survivors in North Carolina, Open Heart, 9, 1, 2022. Crossref

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