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真核基因表达评论综述™
影响因子: 1.660 5年影响因子: 3.082

ISSN 打印: 1045-4403
ISSN 在线: 2162-6502

真核基因表达评论综述™

购买问题 $169.00 卷 9, 1999 册 2

DOI: 10.1615/CritRevEukarGeneExpr.v9.i2

内容列表:

Nuclear Import and Export: Transport Factors, Mechanisms and Regulation
Junona Moroianu
pages 89-106
DOI: 10.1615/CritRevEukarGeneExpr.v9.i2.10
Nuclear snRNA and Nuclear Function (Discovery of 5′ Cap Structures in RNA)
Tae Suk Ro-Choi
pages 107-158
DOI: 10.1615/CritRevEukarGeneExpr.v9.i2.20
Multipotential Cells in the Bone Marrow Stroma: Regulation in the Context of Organ Physiology
Paolo Bianco, Mara Riminucci, Sergei Kuznetsov, Pamela Gehron Robey
pages 159-173
DOI: 10.1615/CritRevEukarGeneExpr.v9.i2.30

真核基因表达评论综述™

Breast Cancer Bone Disease. CRE Vol. 19, Issue 1. Breast Cancer Bone Disease. CRE Vol. 19, Issue 1. Cancer Treatment-Related Bone Disease
The relationship of adipocytes to osetoblasts. Vol. 19, Iss. 2, Marrow Fat and the Bone Microenvironment: Developmental, Functional, and Pathological Implications. The relationship of adipocytes to osetoblasts. Vol. 19, Iss. 2, Marrow Fat and the Bone Microenvironment: Developmental, Functional, and Pathological Implications. Marrow Fat and the Bone Microenvironment: Developmental, Functional, and Pathological Implications
Metallothionein Expression. Vol. 19, Issue 4, Human Metallothionein Expression under Normal and Pathological Conditions: Mechanisms of Gene Regulation Based on In silico Promoter Analysis. Metallothionein Expression. Vol. 19, Issue 4, Human Metallothionein Expression under Normal and Pathological Conditions: Mechanisms of Gene Regulation Based on In silico Promoter Analysis. Human Metallothionein Expression under Normal and Pathological Conditions: Mechanisms of Gene Regulation Based on In silico Promoter Analysis
Schematic structure of mT. Vol.20, Iss. 1, mTOR Signaling in Cancer Cell Motility andtTumor Metastasis. Schematic structure of mT. Vol.20, Iss. 1, mTOR Signaling in Cancer Cell Motility andtTumor Metastasis. mTOR Signaling in Cancer Cell Motility and Tumor Metastasis
Possibilities for how the c-myc proto-oncogene can be amplified in different tumor cells.Vol. 20, Iss. 3, Genome Instability in the Context of ChromatinStructure and Fragile Sites. Possibilities for how the c-myc proto-oncogene can be amplified in different tumor cells.Vol. 20, Iss. 3, Genome Instability in the Context of ChromatinStructure and Fragile Sites. Genome Instability in the Context of Chromatin Structure and Fragile Sites
Schematic representation of the tentative neuroinflammatory mechanism and its relationship to heteroossification. Vol. 20, Iss. 4, Heterotopic Ossification Has Some Nerve. Schematic representation of the tentative neuroinflammatory mechanism and its relationship to heteroossification. Vol. 20, Iss. 4, Heterotopic Ossification Has Some Nerve. Heterotopic Ossification Has Some Nerve